SONG INDUCED GENE EXPRESSION IN THE CANARY BRAIN

degree of confluence. We used RPMI-1640 medium (30 ml for attachedcells; 40 ml for leukaemias) with phenol red, glutamine (2 mM) and 5%fetal calf serum. For compatibility with the drug-profiling regimen, weobtained all fetal calf serum from a large batch (BioWhittaker) used byDTP. No antibiotic[r]

carried out some experiments. SK, RL and JH providedcrucial reagents to carry out the experiments and helped todraft the manuscript. KM participated in the design of thestudy and helped with the confocal microscopy. IJ initi-ated the study, participated i[r]

Germany.7 Riley RL, Blomberg BB & Frasca D (2005) B cells,E2A, and aging. Immunol Rev 205, 30–47.8 Ikawa T, Kawamoto H, Wright LYT & Murre C(2004) Long-term cultured E2A-deficient hematopoieticprogenitor cells are pluripotent. Immunity 20, 349–360.9 Lazorchak AS, Wojciechowski J, Dai[r]

M of methyl viologen. One microgram of RNA was used in each experiment. Samples were collected at the exponential phaseof the PCR. All RT-PCR experiments were repeated twice, and similar results were obtained consistently. Expression of the rnpB gene wasused[r]

2 in response to various external stimuli (Wojtaszek, 1997) therefore their disturbance between production and elimination of the host cell. The decrease in catalytic activity of the plant antioxidant system is also a reason for oxidative stress to appear (Shigeoka[r]

Page 7 of 12Figure 4 Monocyte genes specifically induced by 15 kDa Granulysin. Hierarchical clustering of the 152, 498, 429 and 598 genes induced by15 kDa Granulysin at 4 (a), 12 (b), 24 (c) and 48 (d) hours respectively (p-value < 0.001, FDR < 0.1) and the[r]

expression on human monocytes induced by granulocyte-macrophagecolony-stimulating factor. J Immunol 1993, 150:579-584.14. Hornell TM, Beresford GW, Bushey A, Boss JM, Mellins ED: Regulation of theclass II MHC pathway in primary human monocytes by granulocyte-macrophage colony-st[r]

peat of a DNA sequence by unequal crossing overchorion proteins are also amplified in ovarian follicle(q.v.) or by an accident of replication. Duplicatedcells. Gene amplification can be induced by treatinggenes created in these ways may subsequently evolvecultured cells wit[r]

expression in microglia. Glia 2011, 59:1-13.9. Iadecola C, Salkowski CA, Zhang F, Aber T, Nagayama M, Vogel SN,Ross ME: The transcription factor interferon regulatory factor 1 isexpressed after cerebral ischemia and contributes to ischemic braininjury. J Exp Med 1999, 189:719-72[r]

IRF-2 suppressed the IRF-1-mediated enhancement of gene expression,suggesting that IFN-c-induced L-RAP gene expression is cooperativelyregulated by IRFs and PU.1 transcription factors.AbbreviationsA-LAP, adipocyte-derived leucine aminopeptidase; BAC, bacteri[r]

shows that samples from NHPs exposed to VEEV wereclustered together and maintained a significant distancefrom the control group along first principal componentaxis (x-axis: PCA1), which, incidentally, represents thehighest variance between the two groups.Confirmation of gene [r]

cofactor for MLL-associated leukemogenesis. Cell 123,207–218.16 Hughes CM, Rozenblatt-Rosen O, Milne TA, CopelandTD, Levine SS, Lee JC, Hayes DN, Shanmugam KS,Bhattacharjee A, Biondi CA et al. (2004) Menin associ-ates with a trithorax family histone methyltransferasecomplex and with the hoxc8[r]

F, Murphy BD: Biphasic effects of leptin in porcine granulosacells. Biol Reprod 2003, 68:789-796.37. Jakimiuk AJ, Weitsman SR, Navab A, Magoffin DA: Luteinizing hor-mone receptor, steroidogenic acute regulatory protein, andsteroidogenic enzyme messenger ribonucleic acids are over-expressed