CA125 IN THE PATHOGENESIS OF EPITHELIAL OVARIAN CANCER

analysis. XH performed oophorectomies, ovarian transplants and orthotopicimplantation of tumor cells and AKS conducted the histopathologicalevaluation of tumor tissues. DCC conceived and designed experiments,analyzed the data and wrote the manuscript. All au[r]

In this retrospective study, data from patients listed in the Korea Central Cancer Registry during 1993– 2014 were analysed, to investigate the incidence and survival of second primary cancers (SPCs) after a diagnosis of primary peritoneal, epithelial ovarian, and fallopian tubal (POFT) cancer.

Hadley A, Vinocur JM, Buckanovich RJ, Thompson CB, Levine B, Coukos G:Letal, A tumor-associated NKG2D immunoreceptor ligand, inducesactivation and expansion of effector immune cells. Cancer Biol Ther 2003,2:446-451.80. Connor JP, Felder M: Ascites from epithelial ovarian[r]

Platinum resistance is a major obstacle in the treatment of epithelial ovarian cancer (EOC). Activation of the AKT pathway promotes platinum resistance while inhibition of AKT sensitizes chemoresistant cells. Patients with BRCA mutant EOC, and thus a defect in the homologous recombination (HR) repai[r]

The aim of this systematic review and meta-analysis was to compare overall survival and disease-free survival after fertility sparing surgery (FSS) vs radical surgery in stage 1 epithelial ovarian cancer (EOC).

AB0 blood groups and Rhesus factor expression have been associated with carcinogenesis, response to treatment and tumor progression in several malignancies. The aim of the present study was to test the hypothesis that AB0 blood groups and Rhesus factor expression are associated with clinical outcome[r]

with a favourable prognosis in breast cancer. The aim of this study was to examine the prognostic value of RBM3mRNA and protein expr ession in epithelial ovarian cancer (EOC) and the cisplatin response upon RBM3 depletionin[r]

Muc16 produced by MOVCAR cells. Soluble and cell-associated murine Muc16 were analyzed, respectively, by Western blottingand flow cytometry assays using a new panel of antibodies. The presence of N-linked oligosaccharides on murine Muc16 wasdetermined by ConA chromatography.Resu[r]

Foster R, Dombkowski D, Preffer F, Maclaughlin DT, Donahoe PK: Ovariancancer side population defines cells with stem cell-like characteristicsand Mullerian Inhibiting Substance responsiveness. Proc Natl Acad SciUSA 2006, 103:11154-11159.55. Bapat SA, Mali AM, Koppikar CB, Kurrey NK: Stem and progeni[r]

E, Isselbacher KJ, Wilson JD, Martin JB, Kasper DL, eds.Harrison’s Principles of Internal Medicine. 14th ed. NewYork, NY: McGraw-Hill; 1998:360-362.2. Young RC. Gynecologic malignancies. In: Fauci AS,Braunwald E, Isselbacher KJ, Wilson JD, Martin JB,Kasper DL, eds. Harrison’s Principle[r]

Chapter 093. Gynecologic Malignancies (Part 1) Harrison's Internal Medicine &gt; Chapter 93. Gynecologic Malignancies Ovarian Cancer Incidence and Epidemiology Ovarian cancer can develop from three distinctive cell types (germ cells, stromal cells, and epithelial<[r]

intraperitoneal chemotherapy to treat gastric cancer with ascites and/orperitoneal carcinomatosis: Results from a Chinese center. J Surg Oncol2010, 101:457-464.9. Dovern E, de Hingh IH, Verwaal VJ, van Driel WJ, Nienhuijs SW:Hyperthermic intraperitoneal chemotherapy added to the treatm[r]

The adhesion molecule, FAT4, has a tumor suppressor function with a critical role in the epithelial-tomesenchymal-transition (EMT) and anti-malignant growth in several cancers. No study has investigated yet its role in epithelial ovarian cancer (EOC) progression.

Carcinogenesis by Concurrent Inactivation of p53 and Rb1 in the MouseOvarian Surface Epithelium. Cancer Res 2003, 63:3459-3463.12. Orsulic S, Li Y, Soslow RA, Vitale-Cross LA, Gutkind JS, Varmus HE: Inductionof ovarian cancer by defined multiple genetic chan[r]

We here report our experience on fertility sparing with cervical, endometrial and ovarian cancers and include a review of the literature. With cervical cancer, radical trachelectomy with para-aortic and pelvic lymphadenectomy can be performed in patients with early[r]

carcinoma. Rom J Morphol Embryol 2005, 46:329-334.20. Juretzka MM, Barakat RR, Chi DS, Iasonos A, Dupont J, Abu-Rustum NR,Poynor EA, Aghajanian C, Spriggs D, Hensley ML, Sabbatini P:CA125 levelas a predictor of progression-free survival and overall survival in ovariancancer pati[r]

and there was about a 20 percent chance of cure. With appropriate, with an optimal surgery where you get out all the cancer and giving women intraperitoneal chemotherapy we can push that cure rate very close to about 50 percent. We'd love to have it a hundred, but that's a lot b[r]

H3K27me3 histone marks shape the inhibition of gene transcription. In prostate cancer, the deregulation of H3K27me3 marks might play a role in prostate tumor progression. Our findings point to epigenetic mark H3K27me3 as an important event in prostate carcinogenesis and progression. The results repo[r]

Novel therapeutic approaches are required to treat ovarian cancer and dependency on glycolysis may provide new targets for treatment.

MicroRNA-9 inhibits ovarian cancer cell growth throughregulation of NF-jB1Li-Min Guo*, Yong Pu*, Zhe Han*, Tao Liu, Yi-Xuan Li, Min Liu, Xin Li and Hua TangTianjin Life Science Research Center and Basic Medical School, Tianjin Medical University, ChinaIntroductionOvarian canc[r]

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