BÁO CÁO Y HỌC: " HEPATOPROTECTIVE EFFECTS OF SPIRULINA MAXIMA IN PATIENTS WITH NON-ALCOHOLIC FATTY LIVER DISEASE: A CASE SERIES" PPSX

12 of treatment to document EVR. The decision to continue treatment after week 12 would then be determined based on individual response to treatment (EVR), tolerance to the drugs, laboratory profiles, and the pre-treatment assessment of the severity of the liver disease. During each follow-up, signs[r]

and severity of disease. Am J Gastroenterol 2001; 96:2957. 10. Pagano G, Pacini G, Musso G, et al. Nonalcoholic steatohepatitis, insulin resistance, and metabolic syndrome: Further evidence for an etiologic association. Hepatology 2002; 35:367. 11. Marchesini G, Brizi M, Morselli-Labate AM, et al. A[r]

genotypes 2 and 3 and 48 weeks in genotype 1 infection. Hematologic inclusion criteria included ANC > 800 cells/mL, hemoglobin > 10 g/dL and platelets > 35,000/mL. Growth factors were allowed in the management of therapy-induced cytopenias. The investigators estimated that “one[r]

genotypes 2 and 3 and 48 weeks in genotype 1 infection. Hematologic inclusion criteria included ANC > 800 cells/mL, hemoglobin > 10 g/dL and platelets > 35,000/mL. Growth factors were allowed in the management of therapy-induced cytopenias. The investigators estimated that “one[r]

what appears to be a relatively weak, ineffective immune response since it is inadequate to clear the virus. Since immunologic mediated inflammation is thought to be the mechanism of progressive fibrosis, it is surprising that immune suppression does not ameliorate the disease at the expense[r]

relatively similar pattern to that found in the untreated control cells. The pairwise comparison analysis demonstrated four significantly up-regulated (COBRA1, ITGB4, STAU2, and HMGN3) genes and one down-regulated (ANK3) gene. All these genes exert their function on transcriptional and translational[r]

C. Persistence of hepatitis B and hepatitis C viral genomes in primary liver cancers from HBsAg-negative patients: a study of a low-endemic area. Hepatology 1993;17:20-29. 33. Koike K, Kobayashi M, Gondo M, Hayashi I, Osuga T, Takada S. Hepatitis B virus DNA is frequently found in live[r]

the exact prevalence of which is unknown. Int. J. Med. Sci. 2006, 3 37Chronic hepatitis C In patients with clinical or biological signs of chronic liver disease, chronic hepatitis C is certain when both anti-HCV antibodies and HCV RNA (sought for with a sensitive technique, detecting 50 IU/ml[r]

fat loss rather than diminution in non-fat body mass as indicated by different means: impedance measure-ments, waist, hip and thigh measurements, and sub-cutaneous fat measurements. Earlier, Udani et al. [20] reported reduced body weights and serum triglyc-erides in 14 obese adults receiving 1500 mg[r]

2005;41:289-298. 24. Rodriguez-Luna H, Khatib A, Sharma P, De Petris G, Williams JW, Ortiz J, Hansen K, Mulligan D, Moss A, Douglas DD, Balan V, Rakela J, Vargas HE. Treatment of recurrent hepatitis C infection after liver transplantation with combination of pegylated interferon alpha2[r]

highly significant association between the rare C allele and lower plasma HDL concentrations in female subjects. The effect remained significant after correcting for multiparametric testing according to Bonferoni and was seen only in subjects with a BMI below the median. In addition, the non-[r]

Int. J. Med. Sci. 2006, 3 79International Journal of Medical Sciences ISSN 1449-1907 www.medsci.org 2006 3(2):79-83 ©2006 Ivyspring International Publisher. All rights reserved Review Management of HCV Infection and Liver Transplantation Thomas D. Schiano, and Paul Martin Adult Liver Transplantation[r]

18. Farci P, et al. Hepatitis C virus-associated fulminant hepatic failure. N Engl J Med, 1996. 335(9): 631-4. 19. Vogt M, et al. Prevalence and clinical outcome of hepatitis C infection in children who underwent cardiac surgery before the implementation of blood-donor screening. N Engl J Med, 1999.[r]

and persisted for more than 24 hr. This information provided a novel insight into the mechanisms of action of ethanol (1mM) in human hepatocellular carcinoma cells. Key words: Ethanol, HepG2 cells, protein kinases, signal transduction, transcription factors, gene expression 1. Introduction Ap[r]

6. Knight EL, Verhave JC, Spiegelman D, Hillege HL, de Zeeuw D, Curhan GC, de Jong PE. Factors influencing serum cystatin C levels other than renal function and the impact on renal function measurement. Kidney Int. 2004; 65(4):1416–1421. 7. Shimizu-Tokiwa A, Kobata M, Io H, Kobayashi N, Shou[r]

16. Regelson W, Loria R, Kalimi M. Hormonal intervention: "buffer hormones" or "state dependency". The role of dehydroepian-drosterone (DHEA), thyroid hormone, estrogen and hypophy-sectomy in aging. Ann N Y Acad Sci. 1988; 521: 260-73. 17. Herbert J. Neurosteroids, brain damage, and mental illness.[r]

pattern. It is possible that participants who have been diagnosed with a chronic disease may have changed their diet. To address this concern we adjusted for the presence of chronic disease (CHD, cancer, and stroke) and hypertension throughout the analyses. We also repeated the analyses exclu[r]

Visser R, Harrington M. European study on epidemiology and the management of HCV in the haemodialysis population—Part 1:centre policy. EDTNA ERCA J. 2004; 30(2):84-90. 66. Angelico M, Tisone G, Rapicetta M, Pisani F, Gandin C, Chionne P, Dettori S, Iaria G, Danese V, Orlando G, Casciani CU. Hepatiti[r]

new urate oxidase. [12] In fact, the old and the new urate oxidase do not significantly differ from a pharmacodynamic point of view; the only difference consists in the reduction of rasburicase’s adverse effects. Studies have reported the presence of antibodies antirasburicase in some pa-tien[r]

sity and is influenced by genetic and environmental factors, and in particular, changes in diet and physical activity [8,9]. Lipid abnormalities associated with obe-sity include increased overall production of lipids with elevated concentrations of fatty acids, triacyl-glycerols, and low-density lip[r]