BÁO CÁO Y HỌC: " PHENOTYPIC CHARACTERISTICS OF HUMAN TYPE II ALVEOLAR EPITHELIAL CELLS SUITABLE FOR ANTIGEN PRESENTATION TO T LYMPHOCYTES" PPTX

compared to those with ND. These results are consistent with a previous report among HAART-experienced subjects, thus further implicating HIV DNA in the pathogenesis of HAD. Key words: human immunodeficiency virus type 1; dementia; cognition; HIV DNA 1. INTRODUCTION Complete eradication of the human[r]

outstandingly favorable against H. pylori infections for not interfering the normal bacterial flora in the intestine of humans. Moreover, it should also be advantageous that the compounds of Rice-fluid failed to affect the survival of S. aureus, S. epidermidis, and P. aeruginosa, demonstrating that[r]

Exon 1 (transcript variant 3) and exon 10 were amplified from selected samples with PCR parameters as described above. After a control electrophoresis on 2% agarose gels, the PCR products were directly subjected to processing with Surveyor nuclease [13] (Transgenomic, Omaha, USA) according to the pr[r]

non-normally distributed variables (insulin, HOMA-IR, CRP, adiponectin) for which group median and interquartile ranges are shown. The non-normally distributed variables were log-transformed, checked for normality after log transformation, and used as continuous log-transformed variables for analyse[r]

apoptosis through early phosphorylation of GRK2. Our data correlates with the reported involvement of this protein kinase in the regulation of signal transduction initiated through the GPCR [26, 27]. Furthermore, the regulatory mechanism of ethanol-induced apoptosis through the GPCR signaling pathwa[r]

Int. J. Med. Sci. 2007, 4 30Statistical analysis Data are expressed as mean values ± standard deviation (SD). Results from HepG2 cells treated with 1mM concentration of ethanol (group 1) were compared to non-treated HepG2 cells (control cells, group 2) using Student's t-test. Statistical sig[r]

12. Stuyver L, et al. Hepatitis C virus genotyping by means of 5'-UR/core line probe assays and molecular analysis of untypeable samples. Virus Res 1995, 38: 137-57. 13. Zheng X, et al. Direct comparison of hepatitis C virus genotypes tested by INNO-LiPA HCV II and TRUGENE HCV genotyping methods. J[r]

which is the final irreversible stage characterized by parenchymal nodules with encircling fibrous septa. The hepatitis C virus is not considered to directly injure the liver but it rather triggers an HCV-specific lymphoproliferation. Through profuse cytokine production and also a direct cytopathic[r]

Muscle glycogen Muscle glycogen stores (a surrogate of glucose disposal) were determined by hexokinase enzymatic and spectrophotometric analyses (Sigma Diagnostics, St. Louis, MI) with a C.V. of 5% [21]. Muscle Strength One repetition maximum (1RM) was assessed twice on each machine at baseline (pri[r]

severe with occasional lymphoid aggregates (Figure 2) [4,11]. Actively proliferating bile ductiles are often seen. Risk factors for severe recurrent HCV include advanced donor age, HCV genotype 1, high HCV RNA levels before and after transplant, early histological recurrence of HCV, concomitant cyto[r]

2005; 352(20):2049–2060. 15. Sarnak MJ, Katz R, Stehman-Breen CO, et al. Cystatin C concentration as a risk factor for heart failure in older adults. Ann Intern Med. 2005; 142(7):497–505. 16. Mooradian AD. Digitalis. An update of clinical pharmacokinetics, therapeutic monitoring techniques and treat[r]

antibiotic resistance on the eradication rate of Helicobacter pylori infection by a 1-week regimen of proton pump inhibitor, amoxicillin and clarithromycin. Aliment Pharmacol Ther. 2003; 17: 259-65. 17. Miki I, Aoyama N, Sakai T, et al. Impact of clarithromycin resistence and CYP2C19 genetic[r]

PNA-FISH on metaphase spreads obtained from hESC exposed to A physiological control maintained at 37oC, B 4oC for 24h, C 25oC for 24h, D 4oC for 48h, E 25oC for 48h, The chromosomes were[r]

PPARγ, and PPARδ. While PPARα is mainly expressed in liver, muscle, kidney and heart, PPARγ is most abundant in adipocytes, intestinal cells and macrophages and PPARδ is expressed in many tissues [2-4]. Each subgroup is activated by a certain variety of fatty acids and their derivatives and by speci[r]

severe with occasional lymphoid aggregates (Figure 2) [4,11]. Actively proliferating bile ductiles are often seen. Risk factors for severe recurrent HCV include advanced donor age, HCV genotype 1, high HCV RNA levels before and after transplant, early histological recurrence of HCV, concomitant cyto[r]

with peginterferon alfa-2b plus ribavirin in patients with chronic hepatitis C infected with genotype 1 and low pretreatment viremia. J Hepatol. 2006;44(1):97-103. 10. Drusano GL, Preston SL. A 48-week duration of therapy with pegylated interferon alpha 2b plus ribavirin may be too short to maximize[r]

proteins act downstream of cytokine and growth factor receptors and T- and B-cell antigen receptors. Blood. 1999; 93: 1809-1816. 23. Hamajima N, Matsuo K, Saito T, et al. Interleukin 1 polymorphisms, lifestyle factors, and Helicobacter pylori infection. Jpn J Cancer Res. 2001; 92: 383-[r]

“Ex-smokers” were identified as those who had stopped smoking >1 year before the 1968-1969 examination. Anthropometric measurements in 1968-1969; Body height and body weight were measured with the subjects wearing only briefs. Body mass index (BMI) was calculated by dividing body weight in kg[r]

4. Serfaty L, Aumaitre H, Chazouilleres O, Bonnand AM, Rosmorduc O, Poupon RE, et al. Determinants of outcome of compensated hepatitis C virus-related cirrhosis. Hepatology 1998;27:1435-1440. 5. Kasahara A, Hayashi N, Mochizuki K, Takayanagi M, Yoshioka K, Kakumu S, Iijima A, Urushihara A, Kiyosawa[r]

4. Serfaty L, Aumaitre H, Chazouilleres O, Bonnand AM, Rosmorduc O, Poupon RE, et al. Determinants of outcome of compensated hepatitis C virus-related cirrhosis. Hepatology 1998;27:1435-1440. 5. Kasahara A, Hayashi N, Mochizuki K, Takayanagi M, Yoshioka K, Kakumu S, Iijima A, Urushihara A, Kiyosawa[r]