BÁO CÁO Y HỌC: "EXPRESSION OF AFP AND REV-ERB A/REV-ERB B AND N-COR IN FETAL RAT LIVER, LIVER INJURY AND LIVER REGENERATION" DOC

and persisted for more than 24 hr. This information provided a novel insight into the mechanisms of action of ethanol (1mM) in human hepatocellular carcinoma cells. Key words: Ethanol, HepG2 cells, protein kinases, signal transduction, transcription factors, gene expression 1. Introduction Ap[r]

relatively similar pattern to that found in the untreated control cells. The pairwise comparison analysis demonstrated four significantly up-regulated (COBRA1, ITGB4, STAU2, and HMGN3) genes and one down-regulated (ANK3) gene. All these genes exert their function on transcriptional and translational[r]

and basal core promoter mutations on viral replicative interference. Hepatology 2001;34:404-410. 41. Crespo J, Lozano JL, Carte B, de las Heras B, de la Cruz F, Pons-Romero F. Viral replication in patients with concomitant hepatitis B and C virus infections. Eur J Clin Microbiol[r]

12 of treatment to document EVR. The decision to continue treatment after week 12 would then be determined based on individual response to treatment (EVR), tolerance to the drugs, laboratory profiles, and the pre-treatment assessment of the severity of the liver disease. During each follow-up, signs[r]

20. Hwang SJ, Luo JC, Chu CW, et al. Hepatic steatosis in chronic hepatitis C virus infection: Prevalence and clinical correlation. J Gastroenterol Hepatol 2001;16:190-5. 21. Lonardo A, Adinolfi LE, Loria P, et al. Steatopsis and hepatitis C virus: Mechanisms and significance for hepatic and[r]

2005;41:289-298. 24. Rodriguez-Luna H, Khatib A, Sharma P, De Petris G, Williams JW, Ortiz J, Hansen K, Mulligan D, Moss A, Douglas DD, Balan V, Rakela J, Vargas HE. Treatment of recurrent hepatitis C infection after liver transplantation with combination of pegylated interferon alpha2[r]

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recipients. Clinical Gastroenterol Hepatol. 2005; 10:S125-31 19. Lee W, Dieterich D. Challenges in the management of HIV and hepatitis C virus infection. Drugs. 2004;64(7):693-700 20. Bica I, McGovern B, Dhar R, et al. Increasing mortality due to end-stage liver disease in patients with Human[r]

4. Serfaty L, Aumaitre H, Chazouilleres O, Bonnand AM, Rosmorduc O, Poupon RE, et al. Determinants of outcome of compensated hepatitis C virus-related cirrhosis. Hepatology 1998;27:1435-1440. 5. Kasahara A, Hayashi N, Mochizuki K, Takayanagi M, Yoshioka K, Kakumu S, Iijima A, Ur[r]

Cirrhosis), is a multicenter study of the potential benefit of prolonged peginterferon therapy in mitigating the progression of fibrotic liver disease. [8] In this study, 391 of the 1045 patients enrolled into the initial “lead-in” phase had biopsy proven cirrhosis. Preliminary results show t[r]

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association between hepatitis C infection and survival after orthotopic liver transplantation. Gastroenterology 2002;122:889-96. 10. Berenguer M, Prieto M, San Juan F, Rayon JM, Martinez F, Carrasco D, Moya A, Orbis F, Mir J, Berenguer J. Contribution of donor age to the recent decrease in pa[r]

PPARγ, and PPARδ. While PPARα is mainly expressed in liver, muscle, kidney and heart, PPARγ is most abundant in adipocytes, intestinal cells and macrophages and PPARδ is expressed in many tissues [2-4]. Each subgroup is activated by a certain variety of fatty acids and their derivatives and b[r]

12. Stuyver L, et al. Hepatitis C virus genotyping by means of 5'-UR/core line probe assays and molecular analysis of untypeable samples. Virus Res 1995, 38: 137-57. 13. Zheng X, et al. Direct comparison of hepatitis C virus genotypes tested by INNO-LiPA HCV II and TRUGENE HCV genotyping methods. J[r]

sity and is influenced by genetic and environmental factors, and in particular, changes in diet and physical activity [8,9]. Lipid abnormalities associated with obe-sity include increased overall production of lipids with elevated concentrations of fatty acids, triacyl-glycerols, and low-density lip[r]

oxidase and consequent reduced uric acid concentra-tion. [18] Studies about metabolism have not been per-formed but as other protein, rasburicase metabolism occurs by peptide hydrolysis, so liver should not be involved and the cytochrome P450 is not inducted or inhibited; so even hepatic pathologies[r]

The pShuttle-rBMP4 and pShuttle-rBMP6 plasmids were transfected into 293A cells with Lipofectamine 2000 reagent (Invitrogen, Carlsbad, CA). African green monkey kidney cells (Vero cells, No. CCL-81; American Type Culture Collection [ATCC], Manassas, VA), which have a higher potential t[r]

protein degradation. Metabolism 1997; 46: 673-9. 25. Baulieu EE, Robel P. Dehydroepiandrosterone (DHEA) and de-hydroepiandrosterone sulfate (DHEAS) as neuroactive neuros-teroids. Proc. Natl Acad Sci USA. 1998; 95: 4089-91. 26. Yorek MA, Coppey LJ, Gellett JS, et al. Effect of treatment of diabetic r[r]

were screened for isopropyl thiogalactopyranoside(IPTG)-inducible expression of b-N-acetylglucosamini-dase activity using 4MU-GlcNAc. Five positive clonesCellulomonas fimi b-N-acetylglucosaminidases C. Mayer et al.2930 FEBS Journal 273 (2006) 2929–2941 ª 2006 The Authors J[r]

change in muscle strength (r=0.73, P=0.001). Group assignment was be the only independent predictor of hSGLT3 transcript levels, explaining 68% of its variability (P=0.01). Our data show that hSGLT3, but not GLTU4, expression was enhanced in skeletal muscle after 16 weeks of resistance training. Thi[r]