BÁO CÁO Y HỌC: "CLINICAL ASPECTS AND CYTOKINE RESPONSE IN SEVERE H1N1 INFLUENZA A VIRUS INFECTION" POT
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the HIV-1-infected individuals because no Thai nor-mative data were available to analyze the results. All individuals had minimal/distant or no exposure to illicit drug use with negative urine toxicology screens on two occasions within 30 days prior to enrollment. Subjects were all seronegative for[r]
resistance to some antibiotics. Indeed, H. pylori strains resistant to clarithromycin and metronidazole have been increasing [11-18]. In the near future, antibiotic resistance will be the utmost impediment in the chemotherapy of H. pylori infection. In addition, new triple therapies occasionally cau[r]
and persisted for more than 24 hr. This information provided a novel insight into the mechanisms of action of ethanol (1mM) in human hepatocellular carcinoma cells. Key words: Ethanol, HepG2 cells, protein kinases, signal transduction, transcription factors, gene expression 1. Introduction Ap[r]
oC exposure for 24 h and 48 h). No visible chromosome aberrations (such as fusions and breaks) were detected in the exposed samples. Further analysis of metaphase spreads from different experimental samples using mFISH (24-colour FISH) revealed absence of chromosome translocations demonstrating that[r]
antibiotic resistance on the eradication rate of Helicobacter pylori infection by a 1-week regimen of proton pump inhibitor, amoxicillin and clarithromycin. Aliment Pharmacol Ther. 2003; 17: 259-65. 17. Miki I, Aoyama N, Sakai T, et al. Impact of clarithromycin resistence and CYP2C19 genetic[r]
tions, permitting to continue chemotherapy. [54,55] The use of rasburicase is a good option, sometimes better than use of allopurinol in patients with severe acute hyperuricemia. Allopurinol is a structural analogous of hypoxanthine, inhibitor of xanthine oxi-dase, the last enzyme invo[r]
in this study. There were 4 patients whose gastric at-Int. J. Med. Sci. 2007, 4 100rophy was not examined by the pepsinogen test. The H. pylori positive perticipants were 115 (66.1%) in all patients, and gastric atrophy was observed in 46/170 (27.1%). The gastric atrophy was in 39.1% among 115 posi[r]
was very large. Our original hypothesis was that this would be the case if flavanol-rich cocoa and its influ-ence on nitric oxide synthesis had implications for disease pathogenesis, as has been suggested [26]. We could explain this extraordinary difference in the risk of death by proposing either <[r]
group relegated to the same caloric-restricted diet as the Test group. Many published studies concerning methods to combat obesity provide only scale weight loss as the principal end point. The assumption that scale weight loss is synonymous with fat loss, however, is not al-ways true [26,27]. Scale[r]
were included and 1.2% was found to have severe and 6.2% had mild COPD. 16.1% had only respiratory symptoms at baseline. The survey showed that the presence of obstructive lung disease was a significant predictor of earlier death in long term follow-up. This was true for current and former sm[r]
118mouse embryonic stem (ES) cells carrying targeted mutations of BRCA1 (homozygous deletion of exons 22-24, referred to as BRCA1-/-) or BRCA2 (trEx11/ΔEx27, referred to as BRCA2-/-), Farmer et al. (2005) revealed similar sensitivity of these cells to PARP-1 inhibitors in vitro [25]. Will this work[r]
16. Regelson W, Loria R, Kalimi M. Hormonal intervention: "buffer hormones" or "state dependency". The role of dehydroepian-drosterone (DHEA), thyroid hormone, estrogen and hypophy-sectomy in aging. Ann N Y Acad Sci. 1988; 521: 260-73. 17. Herbert J. Neurosteroids, brain damage, and mental illness.[r]
1 4 1. Department of Hospital Pharmacy, School of Medicine, Kobe University, 7-5-2 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan 2. Clinical Pathology and Immunology, Department of Biomedical Informatics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan 3.[r]
PPARγ, and PPARδ. While PPARα is mainly expressed in liver, muscle, kidney and heart, PPARγ is most abundant in adipocytes, intestinal cells and macrophages and PPARδ is expressed in many tissues [2-4]. Each subgroup is activated by a certain variety of fatty acids and their derivatives and b[r]
Acknowledgements This study was supported by grant RO1 CA070867 from the National Institutes of Health. Conflicts of interest The authors have declared that no conflict of interest exists. References 1. Kawate R, Yamakido M, Nishimoto Y, Bennett PH, Hamman RF, Knowler WC. Diabetes mellitus and its v[r]
Exon 1 (transcript variant 3) and exon 10 were amplified from selected samples with PCR parameters as described above. After a control electrophoresis on 2% agarose gels, the PCR products were directly subjected to processing with Surveyor nuclease [13] (Transgenomic, Omaha, USA) according to[r]
hypothesis and at the surveillance process we study a large number of “exposures” (in this case drugs) and many outcomes (in this case different types of congenital malformations) it is to be expected that a number of apparently significant associations will occur. In this situation th[r]