BÁO CÁO Y HỌC: "THE ROLE OF INTERLEUKIN-1 IN THE PATHOGENESIS OF HUMAN INTERVERTEBRAL DISC DEGENERATION" PDF
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compared to those with ND. These results are consistent with a previous report among HAART-experienced subjects, thus further implicating HIV DNA in the pathogenesis of HAD. Key words: human immunodeficiency virus type 1; dementia; cognition; HIV DNA 1. INTRODUCTION Complete eradicatio[r]
yk23724@gipac.shinshu-u.ac.jp Received: 2006.03.25; Accepted: 2006.06.30; Published: 2006.07.12 Background: Helicobacter pylori has now been widely recognized as a causative agent of gastroduodenal diseases. The development of safer anti- H. pylori compounds is desirable due to the antibiotic-resist[r]
of wild type BRCA1 and BRCA2, DSBs can be efficiently repaired by RAD51 mediated homologous recombination (Fig. 1B). It has been shown that BRCA1 and BRCA2 interact with RAD51, and play essential role in homologous recombination [28, 30, 31]. Thus, in the event of DNA damage associated with P[r]
DHEA-S levels. Additionally, the oldest-old females with lower DHEA-S gained fewer benefits on enhanc-ing motor performance from exercise training. Acknowledgements This research was supported by the National Science Council, ROC, Grant NSC93-2413-H031-004. Conflict of interests The authors have dec[r]
acute HCV infection persists and 4% to 20% of patients with chronic hepatitis C will develop liver cirrhosis within 20 years. In patients with liver cirrhosis, the risk to develop HCC is 1-5% per year. Current standard therapy is the combination of pegylated interferon-α (PEG-IFN-α) and ribav[r]
Corresponding address: Jin Zhong Li, D.V.M. Ph.D., Department of Neurological Surgery, University of Virginia Health System, P. O. Box 800212, Charlottesville, Virginia 22908, USA Received: 2006.05.02; Accepted: 2006.05.31; Published: 2006.06.01 Osteogenic potentials of some recombinant human bone m[r]
and persisted for more than 24 hr. This information provided a novel insight into the mechanisms of action of ethanol (1mM) in human hepatocellular carcinoma cells. Key words: Ethanol, HepG2 cells, protein kinases, signal transduction, transcription factors, gene expression 1. Introduc[r]
randomized study of treatment duration and ribavirin dose. Ann Intern Med 2004;140(5):346-55. 27. Scheuer PJ, Ashrafzadeh P, Sherlock S, et al. The pathology of hepatitis C. Hepatology 1992;15(4):567-71. 28. Sheth SG, Flamm SL, Gordon FD, et al. AST/ALT ratio predicts cirrhosis in patients with chro[r]
with HCV or HBV. However, until now there is no standard of care available for treatment of patients with coinfection. Further clinical trails are needed to clarify the optimal treatment for such patients. Moreover, HCV genotype and HBV genotype were found to be associated with clinical outcome in s[r]
Acknowledgements This study was supported by grant RO1 CA070867 from the National Institutes of Health. Conflicts of interest The authors have declared that no conflict of interest exists. References 1. Kawate R, Yamakido M, Nishimoto Y, Bennett PH, Hamman RF, Knowler WC. Diabetes mellitus an[r]
the MTT assay. The results showed that hESC survivability after exposure to 25oC and 4oC for 24h was 77.3 ± 4.8 % and 64.4 ± 4.4 % respectively (significantly different, P < 0.05). The corresponding survival rates after 48h exposure to 25oC and 4oC was 71.0 ± 0.5 % and 69.0 ± 2.3 % respective[r]
measurement of the oxygen equilibrium curves of Hb J Meerut showed a slightly increased oxygen affinity [4]. In our case, the slight increase in the oxygen affinity of Hb J Meerut relative to that of the normal control has been shown by the low p50 values in arterial whole blood. In Hb J Meerut of g[r]
with peginterferon alfa-2b plus ribavirin in patients with chronic hepatitis C infected with genotype 1 and low pretreatment viremia. J Hepatol. 2006;44(1):97-103. 10. Drusano GL, Preston SL. A 48-week duration of therapy with pegylated interferon alpha 2b plus ribavirin may be too sho[r]
therapy early in the course of HCV. Importantly a predictor of interferon response was an absence of treatment for acute rejection [28]. At our institution, patients with HCV undergo protocol liver biopsies every 6 months, and interferon and ribavirin are started only in the presence of progressive[r]
PPARγ, and PPARδ. While PPARα is mainly expressed in liver, muscle, kidney and heart, PPARγ is most abundant in adipocytes, intestinal cells and macrophages and PPARδ is expressed in many tissues [2-4]. Each subgroup is activated by a certain variety of fatty acids and their derivatives and by speci[r]