BÁO CÁO Y HỌC: ": NETWORK ANALYSIS OF SKIN TUMOR PROGRESSION IDENTIFIES A REWIRED GENETIC ARCHITECTURE AFFECTING INFLAMMATION AND TUMOR SUSCEPTIBILITY" DOCX
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BÁO CÁO Y HỌC: "GENETIC POLYMORPHISMS IN THE NUCLEOTIDE EXCISION REPAIR PATHWAY AND LUNG CANCER RISK: A META-ANALYSIS"
95% confidence interval (CI) = 1.14 - 1.49). We found a protective effect of the XPA 23G/G genotype (OR = 0.75, 95% CI = 0.59 - 0.95). Considering the data available, it can be conjectured that if there is any risk association between a single SNP and lung cancer, the risk fluctuation[r]
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BÁO CÁO Y HỌC: " MUTATION ANALYSIS OF HCDC4 IN AML CELLS IDENTIFIES A NEW INTRONIC POLYMORPHIS"
Int. J. Med. Sci. 2006, 3 148International Journal of Medical Sciences ISSN 1449-1907 www.medsci.org 2006 3(4):148-151 ©2006 Ivyspring International Publisher. All rights reserved Short Research Communication Mutation Analysis of hCDC4 in AML Cells Identifies a New Intronic Polymorphism Dani[r]
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BÁO CÁO Y HỌC: "THE NATURAL HISTORY OF HEPATITIS C VIRUS (HCV) INFECTION"
consumption, age of initial HCV infection, degree of inflammation and fibrosis on liver biopsy, HIV or HBV coinfection, and comordid conditions. An estimated 10%-20% of chronic HCV infections advance to end-stage liver disease over one or two decades. Extrahepatic manifestations can occur during chr[r]
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BÁO CÁO Y HỌC: "PARP-1 INHIBITORS: ARE THEY THE LONG-SOUGHT GENETICALLY SPECIFIC DRUGS FOR BRCA1/2-ASSOCIATED BREAST CANCERS"
mortality in women. Familial breast cancer is a small subset of this disease contributing to about 5 to 10% of breast cancer cases. Germline mutations of the breast tumor suppressor genes BRCA1 and BRCA2 have been found to contribute to most of the familial breast cancer cases [1-5]. BRCA1 wa[r]
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BÁO CÁO Y HỌC ANTIVIRAL THERAPY OF HCV IN THE CIRRHOTIC AND TRANSPLANT CANDIDATE
Cirrhosis), is a multicenter study of the potential benefit of prolonged peginterferon therapy in mitigating the progression of fibrotic liver disease. [8] In this study, 391 of the 1045 patients enrolled into the initial “lead-in” phase had biopsy proven cirrhosis. Preliminary results show t[r]
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BÁO CÁO Y HỌC: " A PRACTICAL APPROACH TO MANAGING PATIENTS WITH HCV INFECTION"
12 of treatment to document EVR. The decision to continue treatment after week 12 would then be determined based on individual response to treatment (EVR), tolerance to the drugs, laboratory profiles, and the pre-treatment assessment of the severity of the liver disease. During each follow-up, signs[r]
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BÁO CÁO Y HỌC: "MALIGNANT PHYLLODES TUMOR WITH HETEROLOGOUS LIPOSARCOMATOUS DIFFERENTIATION AND TUBULAR ADENOMA-LIKE EPITHELIAL COMPONENT"
The epithelial component was of particular interest. It demonstrated a proliferation of moderately dilated compact, rounded regular glands, partially with eosinophilic proteinaceous material, without atypia or mitotic activity, typically seen in a tubular adenoma of the breast (Fig.4,5[r]
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BÁO CÁO Y HỌC: " A DIETARY SUPPLEMENT CONTAINING STANDARDIZED PHASEOLUS VULGARIS EXTRACT INFLUENCES BODY COMPOSITION OF OVERWEIGHT MEN AND WOMEN"
were measured at the beginning and end of the 30-day treatment phase. BMI was calculated as the body weight in kilograms divided by the square of the height in meters. Bodv Weight and Composition Body weights, performed on undressed indi-viduals, were measured using a calibrated Bodymas-ter S[r]
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BÁO CÁO Y HỌC ANTIVIRAL THERAPY OF HCV IN THE CIRRHOTIC AND TRANSPLANT CANDIDATE
Cirrhosis), is a multicenter study of the potential benefit of prolonged peginterferon therapy in mitigating the progression of fibrotic liver disease. [8] In this study, 391 of the 1045 patients enrolled into the initial “lead-in” phase had biopsy proven cirrhosis. Preliminary results show t[r]
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BÁO CÁO Y HỌC: "TREATMENT OF CHRONIC HCV INFECTION IN SPECIAL POPULATIONS."
what appears to be a relatively weak, ineffective immune response since it is inadequate to clear the virus. Since immunologic mediated inflammation is thought to be the mechanism of progressive fibrosis, it is surprising that immune suppression does not ameliorate the disease at the expense[r]
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BÁO CÁO Y HỌC HEPATITIS C VIRUS SEROLOGIC AND VIROLOGIC TESTS AND CLINICAL DIAGNOSIS OF HCVRELATED LIVER DISEASE
the exact prevalence of which is unknown. Int. J. Med. Sci. 2006, 3 37Chronic hepatitis C In patients with clinical or biological signs of chronic liver disease, chronic hepatitis C is certain when both anti-HCV antibodies and HCV RNA (sought for with a sensitive technique, detecting 50 IU/ml[r]
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BÁO CÁO Y HỌC: " LOW CONCENTRATION OF ETHANOL INDUCE APOPTOSIS IN HEPG2 CELLS: ROLE OF VARIOUS SIGNAL TRANSDUCTION PATHWAYS"
and persisted for more than 24 hr. This information provided a novel insight into the mechanisms of action of ethanol (1mM) in human hepatocellular carcinoma cells. Key words: Ethanol, HepG2 cells, protein kinases, signal transduction, transcription factors, gene expression 1. Introduction Ap[r]
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BÁO CÁO Y HỌC: "ASSOCIATION OF THE T+294C POLYMORPHISM IN PPAR Δ WITH LOW HDL CHOLESTEROL AND CORONARY HEART DISEASE RISK IN WOMEN"
Int. J. Med. Sci. 2006, 3 108International Journal of Medical Sciences ISSN 1449-1907 www.medsci.org 2006 3(3):108-111 ©2006 Ivyspring International Publisher. All rights reserved Research paper Association of the T+294C polymorphism in PPAR δ with low HDL cholesterol and coronary heart disease risk[r]
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BÁO CÁO Y HỌC: "RASBURICASE REPRESENTS A NEW TOOL FOR HYPERURICEMIA IN TUMOR LYSIS SYNDROME AND IN GOUT LISA CAMMALLERI AND MARIANO MALAGUARNERA"
Int. J. Med. Sci. 2007, 4 83International Journal of Medical Sciences ISSN 1449-1907 www.medsci.org 2007 4(2):83-93 © Ivyspring International Publisher. All rights reserved Review Rasburicase represents a new tool for hyperuricemia in tumor lysis syn-drome and in gout Lisa Cammalleri and Mar[r]
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BÁO CÁO Y HỌC: "HEPATITIS C VIRUS (HCV) INFECTION AND HEPATIC STEATOSIS"
and severity of disease. Am J Gastroenterol 2001; 96:2957. 10. Pagano G, Pacini G, Musso G, et al. Nonalcoholic steatohepatitis, insulin resistance, and metabolic syndrome: Further evidence for an etiologic association. Hepatology 2002; 35:367. 11. Marchesini G, Brizi M, Morselli-Labate AM, et al. A[r]
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BÁO CÁO Y HỌC: "PROTEOMIC ANALYSIS OF MECHANISMS OF HYPOXIA-INDUCED APOPTOSIS IN TROPHOBLASTIC CELLS"
date the mechanism of this network. However, a poor correlation between mRNA and protein abundance has also been reported [9]. Furthermore, a single gene can encode for more than one mRNA species through differential splicing, and proteins can undergo as many as 200 posttranslational m[r]
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BÁO CÁO Y HỌC: "A POSSIBLE LINK BETWEEN EXERCISE-TRAINING ADAPTATION AND DEHYDROEPIANDROSTERONE SULFATE- AN OLDEST-OLD FEMALE STUDY"
subjects walked briskly for 20 minutes at 45-80% heart rate (HR) reserve (to achieve an intensity of 11-14 of Rating of Perceived Exertion Scale), more than 5 days a week. They also performed a 30 minute resistance training (3 times per week) consisting of a 5-minute warm-up and[r]
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BÁO CÁO Y HỌC: "GRB2-ASSOCIATED BINDER 1 POLYMORPHISM WAS ASSOCIATED WITH THE RISK OF HELICOBACTOR PYLORI INFECTION AND GASTRIC ATROPHY"
is the second most common cause of death from cancer [1]. Helicobacter pylori (H. pylori) strains carrying the cytotoxin-associated gene A (cagA) gene are strongly associated with increased risk of gastric adenocarcinoma [2]. However, only some of those infected developed H. pylori-related di[r]
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BÁO CÁO Y HỌC: "NO INCREASED RISK OF INFANT HYPOSPADIAS AFTER MATERNAL USE OF LORATADINE IN EARLY PREGNANCY"
pregnancy. 3. Results In the repeated analysis of the first period (up to and including 2001) we restricted births to those occurring Int. J. Med. Sci. 2006, 3 107after July 1, 1995 and searched the registers for all known cases of hypospadias. The total number of loratadine-exposed infants is then[r]
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BÁO CÁO Y HỌC: NO ASSOCIATIONS OF HELICOBACTER PYLORI INFECTION AND GASTRIC ATROPHY WITH PLASMA TOTAL HOMOCYSTEINE IN JAPANES
of general populations [8, 9], and as high as 30% of the population aged 65 and older in the Framingham Heart Study (tHcy > 14 nmol/ml) [10]. Increasing tHcy concentrations accelerate cardiovascular diseases by promoting vascular inflammation, endothelial dys-function, and hypercoagulability[r]
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