BÁO CÁO Y HỌC: "INCLUSION OF THE GLUCOCORTICOID RECEPTOR IN A HYPOTHALAMIC PITUITARY ADRENAL AXIS MODEL REVEALS BISTABILITY" PPT
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mitogen-activated protein kinase activation in response to lysophosphatidic acid and epidermal growth factor. J Biol Chem 2000; 275: 13842 – 8. 14. Ymazaki S, Nishida K, Yoshida Y, et al. Gab1 is required for EGF receptor signaling and the transformation by activated ErbB2. Oncogene 2003; 22: 1546-5[r]
and found similar trends. Thus, in this paper we present results that include all self-reported cases of type 2 DM. We excluded women from the current Int. J. Med. Sci. 2006, 3 154analysis who had diabetes or had a positive urinary glucose test at the baseline survey. The total follow-up was[r]
were measured at the beginning and end of the 30-day treatment phase. BMI was calculated as the body weight in kilograms divided by the square of the height in meters. Bodv Weight and Composition Body weights, performed on undressed indi-viduals, were measured using a calibrated Bodymas-ter S[r]
subjects walked briskly for 20 minutes at 45-80% heart rate (HR) reserve (to achieve an intensity of 11-14 of Rating of Perceived Exertion Scale), more than 5 days a week. They also performed a 30 minute resistance training (3 times per week) consisting of a 5-minute warm-up and[r]
glycemic control. Strength training is the most effective lifestyle intervention to increase muscle mass but limited data is available in older adults with diabetes. We determined the influence of strength training on muscle quality (strength per unit of muscle mass), skeletal muscle fiber hypertrop[r]
of wild type BRCA1 and BRCA2, DSBs can be efficiently repaired by RAD51 mediated homologous recombination (Fig. 1B). It has been shown that BRCA1 and BRCA2 interact with RAD51, and play essential role in homologous recombination [28, 30, 31]. Thus, in the event of DNA damage associated with PARP inh[r]
1) Why to Treat HCV Infection The treatment of HCV is justified by the natural history and the outcome of chronic infection and liver injury that is associated with cirrhosis, hepatic decompensation, development of HCC, and mortality. In addition, HCV infection has been linked to a variety of[r]
20. Hwang SJ, Luo JC, Chu CW, et al. Hepatic steatosis in chronic hepatitis C virus infection: Prevalence and clinical correlation. J Gastroenterol Hepatol 2001;16:190-5. 21. Lonardo A, Adinolfi LE, Loria P, et al. Steatopsis and hepatitis C virus: Mechanisms and significance for hepatic and[r]
Kevin Robertson5, Robert Paul6, Cecilia Shikuma1, Victor Valcour1 1. Hawaii AIDS Clinical Research Program, University of Hawaii, Honolulu, HI, USA; 2. Armed Forces Research Inst. Med. Sciences, Bangkok, Thailand; 3. Phramongkutklao Hosp., Bangkok, Thailand; 4. Royal Thai Army Med. Dept., Bangkok, T[r]
non-responders to peginterferon alpha 2b and ribavirin. In: AASLD (Abstract 413); Oct 29- Nov 2 2004; Boston, MA. 36. Gross JB et al. Renew Trial- High dose re-treatment with peginterferon alpha 2b plus ribavirin for non responders in chronic HCV patients. In: AASLD 2003 (Abstract 321); Oct 24-28 20[r]
genotypes 2 and 3 and 48 weeks in genotype 1 infection. Hematologic inclusion criteria included ANC > 800 cells/mL, hemoglobin > 10 g/dL and platelets > 35,000/mL. Growth factors were allowed in the management of therapy-induced cytopenias. The investigators estimated that “one[r]
genotypes 2 and 3 and 48 weeks in genotype 1 infection. Hematologic inclusion criteria included ANC > 800 cells/mL, hemoglobin > 10 g/dL and platelets > 35,000/mL. Growth factors were allowed in the management of therapy-induced cytopenias. The investigators estimated that “one[r]
acute HCV infection persists and 4% to 20% of patients with chronic hepatitis C will develop liver cirrhosis within 20 years. In patients with liver cirrhosis, the risk to develop HCC is 1-5% per year. Current standard therapy is the combination of pegylated interferon-α (PEG-IFN-α) and ribavirin. D[r]
Int. J. Med. Sci. 2006, 3 1114. Skogsberg J, Kannisto R, Roshani L, Gagne E, Hamsten A, Larsson C, Ehrenborg E. Characterization of the human peroxisome proliferator activated receptor delta gene and its expression. Int J Mol Med. 2000; 6: 73-81. 5. Olivier WR, Shenk JL, Snaith MR, Russell CS[r]
. The PCR was con-ducted with initial denaturation at 95°C for 10 minutes, 30 cycles of denaturation at 95°C for 1 minute, an-nealing at 60°C for 1 minute, and extension at 72°C for 1 minute, and a final extension at 72°C for 5 minutes. The primers were F1: 5’-AGC CTC TCC TGA CTG TCA TCC-3’,[r]
served in cod protein-fed and soy protein-fed animals may be due to decreased pancreatic insulin release and/or increased hepatic insulin removal. Recently, Davis et al evaluated effects of casein and soy protein on body weight, plasma cholesterol, and insulin sensi-tivity in male lean SHHF (+/cp) r[r]
at 4°C, washed four times in PBS containing 0.05% Tween-20, and blocked in PBS supplemented with 1% bovine serum albumin for 1 hr at 37°C. Appropriately diluted serum samples were added to antigen-coated plates and incubated overnight at 4°C. Plates were washed four times in PBS–0.05% Tween-20 and i[r]
by all nucleated cells and is secreted into the blood at a constant rate [1, 2]. Cys-C is freely filtered through the normal glomerular membrane and completely reabsorbed, followed by catabolization by the proximal tubular cells [1, 2]. The biological fates of Cys-C are favorable as an endoge[r]